Download Color Atlas of Pediatric Pathology by Aliya N. Husain, J. Thomas Stocker PDF
By Aliya N. Husain, J. Thomas Stocker
Pediatric pathology has been a famous sup-specialty of pathology for nearly twenty years. this day pathology education courses are required to supply schooling in pediatric pathology. as well as pediatric pathology experts, many normal and community-based pathologists usually deal with pediatric specimens. A operating wisdom of present pediatric pathology is key for applicable dealing with of those cases.
The colour Atlas of Pediatric Pathology covers the vast variety of pediatric ailments pathologist will most likely come across and is written through recognized leaders during this box. insurance contains either common and no more more often than not noticeable situations, and every dialogue offers a concise precis of the salient positive aspects of the affliction in addition to expertly chosen, top quality colour photographs. the colour Atlas of Pediatric Pathology is a pragmatic operating source for each pathologist who sees pediatric instances in addition to the pathology trainee. The atlas gains nearly 1,100 high quality photos in addition to very important staging and prognostic (including molecular) parameters.
Features of the colour Atlas of Pediatric Pathology include:
Comprehensive insurance of pediatric surgical and post-mortem pathology
Each bankruptcy is gifted via a well-known expert
Practical presentation: concise textual content highlights diagnostic positive aspects making the atlas an exceptional source for the practitioner
Illustrated all through with nearly 1,100 full-color pictures
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Additional info for Color Atlas of Pediatric Pathology
Note that the inclusions do not enlarge the host nuclei or form cytoplasmic bodies. HSV is a cytodestructive agent that causes foci of coagulative necrosis, as seen in the left side of the photomicrograph. The diagnosis may be confirmed by tissue culture, immunohistochemistry, electron microscopy, or polymerase chain reaction (PCR). Serologic studies may be misleading, because single positive immunoglobulin G (IgG) tests only signify past infection. Sera from immunocompromised patients may show false-negative serological results.
28. Salafia C, Starzyk K, Ossandon M, et al. Lipoprotein (a) deposition in the uteroplacental bed distinguishes normal and preeclamptic pregnancies. Am J Obstet Gynecol. 1996;174:363. 29. Redline RW, Boyd T, Campbell V, et al. Maternal vascular underperfusion: Nosology and reproducibility of placental reaction patterns. Pediatr Dev Pathol. 2004;7:237–249. 30. Morgan T, Craven C, Lalouel JM, et al. Angiotensinogen Thr235 variant is associated with abnormal physiologic change of the uterine spiral arteries in first-trimester decidua.
Bicornuate uterus (B) as well as genitourinary and cardiovascular malformations (not seen here) are common anomalies of internal organs. Cerebral malformations range from arrhinencephaly (absence of olfactory bulbs and tracts) to holoprosencephaly. The latter is seen on coronal sections of the brain as hemispheric fusion with absent corpus callosum (C). Cyclopia and proboscis (D) may also be seen in severe cases. 6 Amniotic band sequence (ABS) occurs in 1 in 1,200–15,000 live births and 1 in 70 still births.