Download Neurotrauma: New Insights into Pathology and Treatment by John T. Weber PDF
By John T. Weber
Neurotrauma is the top explanation for loss of life and incapacity in teens, and the occurrence in older sufferers is expanding. Neurotrauma can be a box in medication with one of many maximum unmet wishes. centred, concentrated and multidisciplinary efforts are required to wrestle this crucial ailment. intriguing findings from simple learn open possibilities for bettering remedy effects. This quantity offers a distinct and accomplished evaluate of the most recent findings and insights on translational learn in neurotrauma. This publication might be a needs to for any TBI or SCI researcher attracted to translating their paintings to the medical institution, in addition to to clinicians attracted to the newest examine findings that could supply novel remedy innovations for his or her sufferers. . Integrates effects from learn on tense mind damage and spinal wire damage, bridging simple technological know-how and medical learn. comprises contributions from around the globe panel of major researchers and clinicians within the fields of TBI and SCI
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Additional resources for Neurotrauma: New Insights into Pathology and Treatment
Sample text
In the acute phase of injury, primary damage occurs as a direct result of a ÃCorresponding author. 1016/S0079-6123(06)61002-9 13 14 Mechanical Input Mechanical Response Injury Response Fig. 1. Steps in CNS trauma. Traumatic brain and spinal cord injuries result from mechanical loading to the tissue. Pathophysiological events are initiated by the mechanical tissue response to impact. insult secondary response lifespan primary response Fig. 2. Temporal aspects of injury. Mechanical loading causes an acute primary phase followed by a prolonged secondary phase.
Taken together, multilevel investigations can be used to eventually decrease the incidence of traumatic CNS injury and improve clinical outcomes. Acknowledgements We acknowledge Liying Zhang and King Yang from Wayne State University for the FEA computer simulations. Partial funding for the results 25 presented was provided by NSF (BES-0093830) and by Cooperative Agreement No. S. Department of Transportation — National Highway Traffic Safety Administration to the University of Alabama at Birmingham, Southern Consortium for Injury Biomechanics.
Shi, R. D. (2002) Pathological changes of isolated spinal cord axons in response to mechanical stretch. Neuroscience, 110: 765–777. Shi, R. and Whitebone, J. (2006) Conduction deficits and membrane disruption of spinal cord axons as a function of magnitude and rate of strain. J. , 95: 3384–3390. A. F. (1997) Characterization of diffuse axonal pathology and selective hippocampal damage following inertial brain trauma in the pig. J. Neuropathol. Exp. , 56: 822–834. M. F. (1999) High tolerance and delayed elastic response of cultured axons to dynamic stretch injury.