Download Robbins and Cotran Atlas of Pathology, 3e by Edward C. Klatt MD PDF
By Edward C. Klatt MD
Designed to counterpoint Robbins and Cotran Pathologic foundation of sickness, 9th Edition and Robbins simple Pathology, 9th Edition, the full-color Robbins and Cotran Atlas of Pathology deals more than 1,500 notable illustrations that vividly depict the commonest ailments lined in pathology classes and USMLE checks. it is a quickly visible reference or evaluation for college students and execs alike.
- Quickly compare gross, microscopic, and radiologic pictures with examples of ordinary organs and tissues.
- Review simply the most important info you want to know with support from huge legends that offer handy summarizations.
- Understand the correlation among pathology and scientific historical past, actual examination findings, and scientific laboratory tests.
- Access the absolutely searchable text online at pupil Consult.
- Visualize key pathologic findings with crystal readability via over four hundred new or up-to-date images.
- Study successfully with this exact significant other product! All chapters were reviewed and revised to mirror the hot content material present in Robbins and Cotran Pathologic foundation of affliction, 9th Edition (ISBN: 978-1-4557-2613-4).
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Extra resources for Robbins and Cotran Atlas of Pathology, 3e
Sample text
The chest wall is normal. C H A P T E R 2 The Heart Figure 2-10 Brown atrophy, gross Virtually all cardiac diseases lead to an enlarged heart. Here is a rare example of brown atrophy in which the heart is small, with chocolate-brown myocardium. In this condition, there is excessive lipochrome (lipofuscin) deposition within the myocardial fibers. Aging and malnutrition may favor this process, a form of cellular autophagocytosis. Antioxidants may protect against such injury. In the normal aging process the amount of lipofuscin increases within myocardial fiber cytoplasm, but not to the degree shown here.
There is progressive right ventricular failure. C H A P T E R 2 The Heart Figure 2-76 Arrhythmogenic right ventricular cardiomyopathy, microscopic There is segmental loss of myocardium in the muscular wall, with replacement by adipose ( ) and fibrous ( ) tissue. Within the fat are strands of residual cardiac myocytes. The subendocardial layer at the right is preserved. This condition is associated with dysrhythmias that can lead to syncope or even sudden death from heart failure. The pathogenesis of this condition involves abnormal desmosome proteins with uncontrolled apoptosis of myocardium, muscular degeneration with fibrofatty replacement, and inflammation.
Another graft extends in a Y fashion just to the right of this to marginal branches of the circumflex artery. A white temporary pacing wire to treat arrhythmias extends from the mid left surface, and a Swan-Ganz catheter that extended to a peripheral pulmonary artery to measure wedge pressure, equivalent to left atrial pressure, emerges from the right atrium. 41 42 C H A P T E R 2 The Heart Figure 2-43 Calcific aortic stenosis, gross An aortic valve need not be bicuspid to calcify. ” Nodules of calcification ( ) are shown on these cusps, as viewed in the aortic outflow tract at the lower left (compare with pulmonic valve at upper right).